Abstract

Health researchers have advocated for a cross-disciplinary approach to the study and prevention of infectious zoonotic diseases, such as Rift Valley Fever. It is believed that this approach can help bring out the social determinants and effects of the zoonotic diseases for the design of appropriate interventions and public health policy. A comprehensive literature review using a systematic search strategy was undertaken to explore the sociocultural and economic factors that influence the transmission and spread of Rift Valley Fever. Although the findings reveal a paucity of social research on Rift Valley Fever, they suggest that livestock sacrificial rituals, food preparation and consumption practices, gender roles, and inadequate resource base for public institutions are the key factors that influence the transmission. It is concluded that there is need for cross-disciplinary studies to increase the understanding of Rift Valley Fever and facilitate appropriate and timely response and mitigation measures.

Highlights

  • Rift Valley Fever (RVF) is an acute viral zoonosis that affects cattle, sheep, and goats and, people and wildlife.[1]

  • The findings reveal a paucity of social research on Rift Valley Fever, they suggest that livestock sacrificial rituals, food preparation and consumption practices, gender roles, and inadequate resource base for public institutions are the key factors that influence the transmission

  • We conducted a search of the PubMed database to identify literature using the term “Rift Valley Fever,” which yielded a total return of 1,321 citations

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Summary

Introduction

Rift Valley Fever (RVF) is an acute viral zoonosis that affects cattle, sheep, and goats and, people and wildlife.[1]. RVF virus (RVFV) infection can occur in humans if they are exposed to the blood, body fluids, or tissues of infected animals. Neutralizing antibodies to RVFV have been shown in wildlife in Kenya, including African buffalo, black rhino, lesser kudu, impala, African elephant, kongoni, and waterbuck. This raises the possibility that wildlife may be reservoirs for the virus during interepidemic periods and play a role in amplifying the virus during epizootics.[4,5,6,7]

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