Abstract
Beta-aminopropionitrile toxicosis in turkeys induced skeletal and cardiovascular lesions. Enlarged hocks with widening of the epiphyseal plate and articular cartilage, green tinting of the skin over swollen joints, and synovitis were the skeletal defects that were found. Joint lesions apparently resulted from impediment of endochondral mineralization and maturation of chondrocytes in the epiphyseal plate and articular cartilage. Cardiovascular alterations resulting from BAPN feeding included ground substance increase, swelling and disorganization of collagenous fibers, and elastolysis. These changes cause polypoid intimal thickenings and the induction of dissecting aneurysms. Intimal arteriosclerotic plaques were found in the posterior aorta of “normal” turkey poults. There is a possibility that such lesions are a prerequisite for the development of dissecting aneurysms.
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