Abstract
The increase in diffusion length of oxygen in the proliferating tissues including neointima and atherosclerosis may cause a relatively hypoxic state which in turn triggers neovascularization by the production of various angiogenic factors [1]. The inhibition of neovascularization in vascular walls is associated with the attenuated neointima formation [2], which raises the possibility that angiogenesis in vessel walls is involved in the development of neointima.
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