Abstract
Retinoic acid (RA) may promote cell survival or apoptosis depending on the ratio of the ligand binding proteins: apoptosis-inducing cellular RA binding protein 2 (CRABP2), and cell survival-promoting fatty acid binding protein 5 (FABP5). Decreased uptake of retinol by stimulated by RA 6 (STRA6) and altered actions of RA related to reduced expression of CRABP2 may contribute to the development of endometriosis. Statins inhibit growth of human endometrial stromal (HES) cells and reduce endometriotic implants in the rodent models of disease.
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