Abstract
increased alter PVL in the absence of indomethacin treatment [n contrast, PSS was elevated in both PVL and PVL-indo when compared to sham and sham-indo groups. To delineate the isoinrm of cyclooxygenase involved, Cox-1 -A and Cox-2 4mice were sutNected to sham in=4) or PVL in=7) and after 7 days SPP, abdominal aortic flow and PSS were evaluated Contrary to the pharmacological blockade of Cox, the targeted deletinn ot the Cox1 or Co>2 isoforms in isolation was not able to abrogate the devated hemodynamms observed with experimental PHT.
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