Abstract
The acid secretory response of the Heidenhain pouch to food was reduced in all animals who had vagal denervation of the antral mucosa. No difference was noted between vagal denervation by seromuscular transection and antroneurolysis on the amount of depression produced on pouch secretion. Vagal-induced antral motility was unchanged by antroneurolysis. Seromuscular transection abolished vagal-induced antral motility. After ingestion of food, the antrum in situ vagal influence on gastrin release accounted for 30 per cent or more of the Heidenhain pouch response, 40 per cent or more was obtained from mechanical and chemical mechanisms, and 5 per cent or more was of intestinal origin. It is concluded that in the dog the in situ vagal component of gastrin release is significant.
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
