Abstract
Background Chronic hyperglycemia resulting from diabetes mellitus undertakes several body cells through the formation of advanced glycation end products and activation of the polyol pathway (Brownlle, 2005), promoting reduction of electrical activity in skeletal muscle and the regenerative capacity of its cells, as well as impaired contractility and increased tissue stiffness. In turn, the amplify muscle work during exercise is able to elicit several essential biochemical reactions for hypertrophy, power gain and enhance muscle function (Andersen et al., 1996, 1997, 2004, 2005).
Highlights
Chronic hyperglycemia resulting from diabetes mellitus undertakes several body cells through the formation of advanced glycation end products and activation of the polyol pathway (Brownlle, 2005), promoting reduction of electrical activity in skeletal muscle and the regenerative capacity of its cells, as well as impaired contractility and increased tissue stiffness
gastrocnemius-plant complex demonstrated in diabetic animals (GDS) and GDT animals were administered with an intraperitoneal dose of streptozotocin to induce diabetes, which was confirmed by measurement of blood glucose fasting on the 3rd and 7th day post induction
GCT and GDT animals were submitted to an overload jumping exercise program of up to 50% body weight, 5 times a week during 9 weeks
Summary
Chronic hyperglycemia resulting from diabetes mellitus undertakes several body cells through the formation of advanced glycation end products and activation of the polyol pathway (Brownlle, 2005), promoting reduction of electrical activity in skeletal muscle and the regenerative capacity of its cells, as well as impaired contractility and increased tissue stiffness. The amplify muscle work during exercise is able to elicit several essential biochemical reactions for hypertrophy, power gain and enhance muscle function (Andersen et al, 1996, 1997, 2004, 2005)
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