Abstract
s S77 (2,7-fold) or transferred to hypoxic condition (1,3-fold) (p 2,7 fold at 10E-6M, p 2 fold) of more than 100 genes indicating improvement of stem cell paracrine potential. CONCLUSION: Celastrol is involved in multiple survival pathways and a short burst treatment is associated with better survival in oxidative and hypoxic stress in a dose dependent manner. Short burst low-dose treatment with Celastrol may promote MSC scalability without altering stem cell phenotype, differentiation potential and functionality. Together, these results suggest that Celastrol conditioning could be efficient for enhancing stem cell therapeutics for our next generation clinical experiments. FRQS Canadian Cardiovascular Society (CCS) Oral CANADIAN ADVANCES IN VASCULAR DISEASE Saturday, October 24, 2015 150 SEX DIFFERENCES IN THE LONG-TERM OUTCOMES OF PERIPHERAL ARTERIAL DISEASE MA Hussain, T Lindsay, M Mamdani, X Wang, S Verma, M Al-Omran
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