Abstract
Rationale: Selenium plays a pivotal role in the endogenous antioxidant defense mechanism and inflammatory pathways. Previous studies reported that most of critically ill children have low serum selenium levels upon the pediatric intensive care unit (PICU) admission time. Decreased serum levels of selenium were associated with increased incidence of multiple organ failure and deteriorated clinical outcomes. There is an obvious lack of evidence investigating the acute changes of serum selenium concentrations following acute stress.
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