Abstract
Background and Aims : Osteocalcin (OC), an osteoblast-derived regulator of metabolic processes, and circulating early endothelial progenitor cells (EPC, CD34−/CD133+/KDR+) expressing OC (OC+) may represent a pathophysiological link between bone metabolism and the vasculature and be involved in the pathophysiology of vascular atherosclerotic calcification. This study assessed the association of plasma levels of different OC forms and of EPC-OC+ count with the severity of disease in patients with documented coronary atherosclerosis (CAD).
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