Abstract
Systemic sclerosis (SSc) is a multisystem connective tissue disease characterized by immune abnormalities, vasculopathy and fibrosis of the skin and certain internal organs. Recent evidence suggests the potential contribution of a dysregulated innate immune system to its pathogenesis [1]. Mannose-binding lectin (MBL), a member of the collectin family produced mainly by liver hepatocytes and, to a smaller extent, by gastrointestinal and reproductive tracts, binds to various pathogens as a pattern-recognition [...]
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