Serum cardiac markers response to biphasic compared with monophasic electrical cardioversion

R Skulec,J Belohlavek,M Aschermann,T Kovarnik,M Psenicka,A Linhart

doi:10.1186/cc1951

Abstract

Defibrilation or cardioversion in critically ill cardiac patients may be followed by serum cardiac marker elevation. However, only few studies with limited patient samples assessing electrical myocardial injury have been published [1,2]. The aim of our study was to evaluate the response of serum cardiac markers following elective cardioversion for supraventricular tachyarrhyth-mias (SVT) and to analyze the impact of type of shock waveform.

Highlights

Activation of the HPA axis occurs in order to control potentially deleterious effects of systemic inflammation during sepsis
A recent study showed that vascular endothelial damage was the primary cause of multiple organ failure (MOF) in patients with thrombopenia and that humoral mediators played a major role in the development of this process [1]
We aimed to evaluate the relation between the platelet counts (PC), the C-reactive protein and plasma fibrinolytic capacity in intensive care unit (ICU) patients

Summary

Introduction

Activation of the HPA axis occurs in order to control potentially deleterious effects of systemic inflammation during sepsis. The aim of the study was to evaluate the effects of PEEP on intrathoracic blood volumes and cardiac function measured by the COLD system in ARDS patients. The objective was to identify risk factors and outcome of pregnant women who required intensive care This pilot study aims to find stress (S) and other personality traits (physical symptoms [PS], anger [A]) among the ICU staff, and any existing correlation between them and length of working time (LWT) in the ICU and with educational level (EDL). The role of Drotrecogin alfa (activated) (recombinant human activated protein C [rhAPC]) in modulating microvascular coagulation through the inhibition of thrombin generation has been well studied in experimental and clinical settings of severe sepsis, little is known about its direct anti-inflammatory effects on vascular endothelial cells. The aim is to determine whether an effective lifestyle adaptation program post CABG could enhance the quality of life of the CABG patient

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