Abstract
It is approximately 20 years since the serotonin (5-hydroxytryptamine; 5-HT) hypothesis of appetite control was formally stated. In that time, evidence has accumulated to confirm the role of serotonergic mechanisms in appetite control. At present, it is believed that serotonin 5-HT1B and 5-HT2C receptor subtypes mediate the capacity for an inhibition of food intake. Animal studies show that serotonin-induced suppression of eating generally preserves the behavioural satiety sequence, which is widely regarded as an indication of the operation of the natural physiological processes for meal termination and sustained post-meal satiety.
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