Abstract
Submit Manuscript | http://medcraveonline.com transients recorded with the Ca 2+ probe aequorin during isometric contraction of myocardium in patients with end-stage failure were markedly prolonged, with a peak and then a secondary peak in the [Ca 2+ ]i transient trace, which was associated with a marked prolongation of the time course of the Ca 2+ decline and of tension decline [2]. Because SERCA is responsible for 60% to 90% of the [Ca 2+ ]i decline in mammalian ventricular myocytes, these results were consistent with impaired function of the SR Ca 2+ transport
Sign-in/Register to access full text options 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
