Abstract

Self-reported occupational noise exposure has been associated with impaired hearing, but its relationship with extra-auditory affects remains uncertain. This research assessed the association between self-reported occupational noise exposure and cardiovascular outcomes. Participants (n = 6318, ∼50% male) from the Canadian Health Measures Survey (2012-2015) aged 20-79 years were randomly recruited across Canada. An in-person household interview included basic demographics, perceived stress, diagnosed health conditions, and self-reported exposure to a noisy work environment. Direct physiological assessment in a mobile examination centre permitted the determination of biomarkers/risk factors related to cardiovascular function. Logistic or linear regression models explored the association between self-reported occupational noise exposure and several cardiovascular endpoints after adjusting for confounding variables. After adjustments, there was no evidence for an association between occupational noise and any of the evaluated endpoints, which included but were not limited to blood pressure, heart rate, blood glucose, insulin, lipids, diagnosed hypertension, medication for hypertension, myocardial infarction, stroke, or heart disease. There was no evidence that self-reported occupational noise exposure was associated with evaluated cardiovascular-related biomarkers, or cardiovascular diseases among Canadians aged 20-79 years. This study, and others like it, provides an important contribution to an evidence base that could inform policy related to occupational noise exposure.

Highlights

  • The exposure and response to chronic stressors can lead to various illnesses broadly categorized as stress-related health effects (Anisman, 2015; Yusuf et al, 2004)

  • In the current analysis we report the association between selfreported occupational noise exposure and several outcomes including, but not limited to, measured blood pressure, heart rate, blood lipids, glucose, insulin, high sensitivity Creactive protein (HS CRP), apolipoprotein (APO) A/B1, medically diagnosed history of myocardial infarction, heart disease, stroke, hypertension, and self-reported medication usage

  • Among the medically diagnosed conditions considered in this analysis a higher prevalence of participants with hearing impairment, diabetes, high blood pressure, heart disease, myocardial infarction, and taking medication for high blood pressure was observed among those who worked in a noisy environment for 10 years or more (Table I)

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Summary

INTRODUCTION

Gopinath et al, 2011; Kerns et al, 2018; Skogstad et al, 2016; Zhou et al, 2019) possibly owing to higher noise levels and more precisely defined exposures Based on their analysis of the data collected in the National Health and Nutrition Examination Survey (NHANES), Gan et al (2011) reported increased odds of angina pectoris, coronary artery disease and isolated diastolic hypertension (IDH) among a nationally representative sample of workers that self-reported exposure to loud occupational noise. These associations remained after adjusting for several covariates and were more profound when the analysis was restricted to respondents below 50 years of age. In the current analysis we report the association between selfreported occupational noise exposure and several outcomes including, but not limited to, measured blood pressure, heart rate, blood lipids, glucose, insulin, high sensitivity Creactive protein (HS CRP), apolipoprotein (APO) A/B1, medically diagnosed history of myocardial infarction, heart disease, stroke, hypertension, and self-reported medication usage

METHODS
Study sample
Occupational noise exposure assessment
Evaluated outcomes
Covariates
Statistical analysis
RESULTS
DISCUSSION
Full Text
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