Abstract
Background: Alterations in glucose and lipid metabolism in the setting of insulin resistance (IR) play an important role in the progression from Non-alcoholic Fatty Liver (NAFL) to Non-alcoholic Steatohepatitis (NASH) via oxidative stress. Selenoprotein P (SeP) is a selenium carrier protein with antioxidant properties that is higher in obese and diabetic subjects but its potential role as biomarker of NASH is not fully elucidated yet.
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