Abstract
Longterm results of surgical vessel reconstruction are compromised by restenosis due to neointimal hyperplasia. Recent studies suggest that reduced cyclic guanosine monophosphate-signaling is associated with neointima formation. In a rat model of endarterectomy, we investigated the effect of pharmacological inhibition of cyclic guanosine monophosphate-degradation on neointima formation by using the selective phosphodiesterase-5-inhibitor vardenafil.
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