Abstract
Selection of potato cyst-nematode populations of several pathotypes on resistant Solanum vernei hybrids resulted in increased virulence in some populations. This increase is believed to be due to the selection of specific virulence genes in each population gene pool. Resistance based on S. vernei is race specific and its life in the field will probably be finite. Populations of Globodera pallida are conventionally classified into different pathotypes according to their response on resistant test plants. However, populations of the same pathotype did not all behave identically. Such variation of virulence within a pathotype suggests different complements and frequencies of genes for virulence. Many field populations may not fit simply into the current pathotype scheme but no alternative is at present available.
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