Abstract
Background: miRNAs (miR) are critical regulators of the immune system. Recently they have been shown to be secreted into EVs for inter-cell communication (Valadi et al., Nat Cell Biol, 2007). We previously identified an up-regulation of miR-17 and -21 in murine lung homogenate in Ovalbumin (OVA) induced allergic airway inflammation (AAI).
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