Abstract
125 ineglect (p = .003). These findings suggest that in humans as in rodents, ischemic insults to antero-lateral cortex may be a not uncommon antecedent of striatal DA hyperactivity. We hypothesize that penetrance of some common psychosis genes may be variable because these genes merely predispose carriers to responding to minor cortical ischemia with a marked subcortical DA supersensitivity. Other data in support of this hypothesis will be reviewed. (Supported by the VA and R29-MH43530-01)
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