Abstract
Mitochondrial dysfunction is involved in the pathogenesis of diabetic kidney disease. Mitochondrial quality control is characterized by repairing of mitochondrial damage through mitophagy and fission/fusion. It has been shown that blockade of KCa3.1, a potassium channel, ameliorates diabetic renal fibrosis. However, the role of KCa3.1 in mitochondrial quality control is not yet known. The aim of the study is to identify the role of KCa3.1 in mitochondrial quality control in diabetic kidney disease.
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