Abstract
Normal pregnancy is characterized by a ~40% increase in blood volume and cardiac output, mild tachycardia, and decreased arterial blood pressure due to a substantial decrease in total peripheral resistance (TPR). Increased susceptibility to orthostatic and hemorrhagic hypotension is significant problems in term pregnant women and animals, and a number of studies suggest that arterial baroreflex compensatory responses are suppressed in pregnancy. In near term pregnant rats and rabbits, maximum gain for arterial baroreflex control of renal sympathetic nerve activity (RSNA) is decreased, largely due to an attenuated ability to increase RSNA in response to a hypotensive challenge.
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