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Abstract
manic-like behavior. In addition, we evidenced that SD decreased cell proliferation in the dentate gyrus. Interestingly, this effect was acutely rescued by both PKC inhibitors. Taken together, our results support the hypothesis that an overactivation of the PKC signaling system may be crucial for the deficits of neuroplasticity associated with bipolar disorder. Overall, these findings may provide new insights on the pathophysiology of mania and the use of PKC inhibitors as possible novel treatments for mood disorders.
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