Abstract
Hepatitis B X-interacting protein (HBXIP) was named for its interaction with the hepatitis B virus X protein. HBXIP can affect cell proliferation, migration, invasion, angiogenesis, apoptosis, metabolism, and immune escape, it is involved in the regulation of oncogene expression and multiple cell signaling pathways, playing critical role in tumorigenesis and development. For several years, our group has conducted research on the role and mechanism of HBXIP in cancer. This review summarizes the various roles of HBXIP in tumor development and associated molecular mechanisms.
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