Abstract
Until date, diabetes during pregnancy has been linked to an increased risk of maternal, fetal, and neonatal morbidity and mortality. Infants of diabetic mothers (IDM) frequently have difficulties related to fetal hyperglycemia and hyperinsulinemia caused by maternal hyperglycemia. Because insulin breakdown is accelerated in the presence of mild hemolysis, cord serum C-peptide levels are utilized as an indicator of fetal beta-cell function rather than insulin levels. Objective:
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