Abstract
Myelination is an essential feature of the vertebrate nervous system that provides electrical insulation to axons, thereby facilitating the transmission of nerve impulses. Deficiencies in myelination in diseases such as multiple sclerosis (MS) lead to serious neurological disorders. Most MS patients initially exhibit a relapsing-remitting disease course that eventually converts to a secondary progressive form of the disease with incomplete recovery. Therefore, it is important to elucidate not only the causal reason for failure to remyelinate, but also the intrinsic regulatory mechanism(s) underlying successful myelination in the central nervous system (CNS).
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