Abstract
Study Objective: Natural killer (NK) lymphocytes are central to innate immunity and contribute to tissue homeostasis by eliminating altered cells. Their NKG2D receptor pathway plays a fundamental role in target elimination through binding NKG2D ligands on the cell surface. Reduction in the NKG2D ligand, ULBP2, expression is associated with immune resistance in neoplastic processes. We have shown that fibroblasts from adhesion tissue (AT) are characterized by increased extracellular matrix molecules and inflammatory cytokines compared with normal peritoneal (NP) fibroblasts.
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