Abstract
Injection of cortisone into mice simultaneously with typhoid endotoxin prevents the decrease in tryptophan oxygenase activity in the liver and prevents death of the animals. Tryptophan also maintained a normal level of activity of the enzyme but did not prevent death of the animals poisoned with endotoxin. The role of the level of tryptophan oxygenase activity in the pathogenesis of poisoning produced by endotoxin is discussed.
Full Text 
Sign-in/Register to access full text options
Sign-in/Register to access full text options 
Paper version not known
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a call
