Abstract
Nutritional stress applied prior to UV-irradiation to E. coli 15 555-7 reduced thymine dimer excision and inhibited post-UV incorporation of thymidine in polB + as well as in polB − cells. However, the pre-UV-stressed polB + cells were significantly more UV-resistant and after UV synthesized larger DNA molecules than the pre-UV-stressed polB − cells. The data suggest that DNA polymerase II is involved in the tolerance of unremoved thymine dimers.
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Schedule a callMore From: Journal of Photochemistry and Photobiology B: Biology
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