Abstract
These results suggest that 1) glyceollin biosynthesis may not be solely dependent on the induction of enzymes of early phenylpropanoid and flavonoid metabolism in this organ and 2) resistance to P. m. f. sp. glycinea race 1, as defined by the Rps 1 c gene in this organ, may reside partly in the rapid release of the isoflavone aglycones from their conjugates and/or in the later steps of glyceollin biosynthesis
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