Abstract
Abnormal brain excitability as a migraine trigger mechanism, and the use of antiepileptic drugs (AED) in migraine prevention, are reviewed from The Chicago Medical School, North Chicago, IL.
Highlights
Symptoms and neurocognitive functioning were compared in athletes with no headache, athletes complaining of HA (HA group), and athletes with posttraumatic migraine (PTM group), in a study of 261 high school and college athletes at University of Pittsburgh Medical Center; University of North Carolina, Chapel Hill; and Florida Neuroscience Institute, Orlando
Brain imaging with functional MRI and magnetoencephalography (MEG) have shown that the migraine aura is linked with an abnormal electric and metabolic event consistent with the cortical spreading depression of Leao
Visual activation monitored by MEG and fMRI confirm hyperexcitability of the occipital cortex with triggering of the migraine aura
Summary
Symptoms and neurocognitive functioning were compared in athletes with no headache (non-HA group), athletes complaining of HA (HA group), and athletes with posttraumatic migraine (PTM group), in a study of 261 high school and college athletes (mean age, 16.36+/-2.6 years) at University of Pittsburgh Medical Center; University of North Carolina, Chapel Hill; and Florida Neuroscience Institute, Orlando. ROLE OF ANTIEPILEPTIC DRUGS IN MIGRAINE PREVENTION Abnormal brain excitability as a migraine trigger mechanism, and the use of antiepileptic drugs (AED) in migraine prevention, are reviewed from The Chicago Medical School, North Chicago, IL.
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