Abstract
Vitiligo is an acquired pigmentary disorder characterized by the development of depigmented lesions in a variable distribution, owing to the loss or destruction of functioning melanocytes (Guerra et al., 2010). Complex interactions between genetic, immunological, biochemical, and environmental factors are likely to be related to the development of vitiligo (Malhotra and Dytoc, 2013). Despite having identified multiple risk factors of vitiligo, the molecular mechanisms of its pathogenesis remain obscure.
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