Abstract
CT and MR patterns of focal perfusion and diffusion disorder are compared in groups of patients with nonhemorrhagic venous and ischemic stroke, as well as in control group with chronic brain ischemia. The rMTT(+ vs. ++), rCBF (± vs. – –), rCBV (+ vs. ±), rDWI (± vs. +), and rADC (+ vs. –) patterns are determined as reliably distinguishable ( p < 0.05), which is indicative of congestion in venous stroke focus as a releaser of pathophysiological mechanism of development of secondary ischemia.
Highlights
Сравнены КТ- и МР-паттерны фокального нарушения перфузии и диффузии в группах пациентов с негеморрагическим венозным и ишемическим инсультом, а также в контрольной группе с ХИГМ
CT and MR patterns of focal perfusion and diffusion disorder are compared in groups of patients with nonhemorrhagic venous and ischemic stroke, as well as in control group with chronic brain ischemia
The rMTT(+ vs. ++), rCBF, rCBV (+ vs. ), rDWI, and rADC (+ vs. –) patterns are determined as reliably distinguishable (p < 0.05), which is indicative of congestion in venous stroke focus as a releaser of pathophysiological mechanism of development of secondary ischemia
Summary
Семёнов С.Е., Шатохина М.Г., Нехорошева А.Н., Хромов А.А., Жучкова Е.А., Молдавская И.В., Портнов Ю.М., Коваленко А.В., Барбараш Л.С. Role and CT and MR signs of focal congestion in development and course of stroke Semyonov S.Ye., Shatokhina M.G., Nekhorosheva A.N., Khromov A.A., Zhuchkova Ye.A., Moldavskaya I.V., Portnov Yu.M., Kovalenko A.V., Barbarash L.S. Семёнов С.Е., Шатохина М.Г., Нехорошева А.Н. Достоверно различными (p < 0,05) определены паттерны rMTT(+ vs ++), rCBF ( vs – –), rCBV (+ vs ), rDWI ( vs +) и rADC (+ vs –), что свидетельствует о полнокровии в очаге венозного инсульта как пусковом факторе патофизиологического механизма развития вторичной ишемии. The rMTT(+ vs ++), rCBF ( vs – –), rCBV (+ vs ), rDWI ( vs +), and rADC (+ vs –) patterns are determined as reliably distinguishable (p < 0.05), which is indicative of congestion in venous stroke focus as a releaser of pathophysiological mechanism of development of secondary ischemia. Цель исследования — определение КТ- и МРпаттернов фокального нарушения перфузии и диффузии при ВИ и связи их с течением заболевания
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