Abstract
Background: Prostate carcinoma (PCa) is the second most common malignant disease in men. Despite evidence that prostate-specific antigen (PSA) screening can reduce PCa specific metastasis and death, it has not accepted by various health authorities. In fact, a broad range of heterogeneity causes different clinical and molecular behavior of PCa; risk stratification thus is helpful in guiding the optimal treatment of PCa patients.
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