Abstract
Helicobacter pylori infection increases the risk for gastric cancer. Evidence suggests that this risk persists after H. pylori eradication. To identify possible histologic and endoscopic factors associated with increased risk in this setting, investigators in Japan followed a cohort of 573 patients with successful H. pylori eradication for a mean of 6.2 years. All patients had the location of histologic intestinal metaplasia (IM) and the location and severity of endoscopic gastric atrophy …
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