Abstract
Introduction: Weight loss surgery has become an increasingly popular means of combating the obesity epidemic in modern society but like any procedure, it does not shy away from immediate and long-term complications. Copper deficiency has occasionally been reported to occur many years afterwards but with an increased incidence of bariatric procedures and reduced awareness, the effects of this deficiency could now appear to favor an earlier onset. Case Report: We report a case of a 56-year-old Caucasian female with a history of gastric bypass surgery five year ago; with an unsteady gait, weakness, decreased visual acuity, tingling with numbness in her hands and pancytopenia for the last month. She was treated for copper deficiency. Conclusion: Effects of copper deficiency have been shown to cause a wide array of abnormalities related to inactivation of enzymes such as cytochrome c oxidase, superoxide dismutase, dopamine beta hydroxylase and metallothionein. This can lead to reduced nerve transmission within the central nervous system causing motor and sensory polymyeloneuropathy and an overall reduction of energy required for blood cell formation. With early surveillance, such anomalies can be detected and potentially reverse the effects of this micronutrient deficiency.
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