Abstract
We describe a patient with MELAS (mitochondrial myopathy, encephalopathy, lactic acidosis, and stroke-like episodes) syndrome and frequent brief partial seizures arising from the occipital lobe. After a cluster of elementary and complex partial seizures, the patient developed significant reversible focal occipital brain edema. We suggest that the pathogenesis of this prominent edema is a consequence of chronically decreased nucleotide levels in the brain, leading to an altered influence of excitatory amino acid neurotransmitters on membrane ion flux.
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