Abstract
In the setting of systolic heart failure (sHF), left ventricular reverse remodelling (LVRR) has been regarded as an auspicious sign associated with better outcomes [1]. LVRR is generally defined as a gradual improvement in left ventricular systolic functions as measured with a significant and time-dependent increase in left ventricular ejection fraction (LVEF) along with a reduction in left ventricular volumes in patients with sHF [2]. In patients with sHF, even though LVRR might occur spontaneously (under optimal medical therapy) [1], it is more likely to emerge after relief of underlying hemodynamic stress including correction of aortic valvular regurgitation [2,3] or initiation of certain invasive therapeutic regimens including cardiac resynchronization therapy (CRT) [4].
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