Abstract
Right ventricular failure (RVF) represents a formidable clinical challenge that carries significant morbidity and mortality [1]. The management is mainly focused on the treatment of the underlying condition, with additional interventions aimed at optimizing preload, afterload, and contractility [1,2]. However, there is a lack of therapies that specifically target the dysfunctional right ventricle (RV). Recent studies have identified the importance of interventricular interactions, right and left ventricular (LV) dyssynchrony, and subsequent impairment of LV function in the setting of RVF [3–5].
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