Abstract

Abstract Fruit maturity, diphenylamine (DPA) treatment and controlled atmosphere (CA) storage regimes were evaluated as factors affecting development of ‘Fuji’ apple CO 2 injury. The incidence and severity of CO 2 injury (brown-heart) increased with advanced maturity and greater watercore severity at harvest. The development of CO 2 injury during storage was prevented by DPA treatment. The severity of CO 2 injury was higher in fruit exposed to 20 kPa CO 2 after harvest than in fruit exposed to 20 kPa CO 2 after 8 months of CA storage. Ethanol, acetaldehyde and methanol concentrations increased during short- and long-term exposure to high CO 2 , however, DPA reduced ethanol and acetaldehyde accumulation. Storage in low O 2 (0.5 kPa O 2 , 0.05 kPa CO 2 ) also stimulated accumulation of ethanol, acetaldehyde and methanol however, no internal disorders developed with or without the use of DPA. The harvest date effect on severity of CO 2 -injury following short-term exposure to 20 kPa CO 2 was related to severity of CO 2 -injury after long-term CA storage with 3 kPa CO 2 .

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