Abstract
The hand-arm vibration syndrome (HAVS) contains both vascular (vibration induced white finger (VIWF)) and sensorineural components. Although a close link between the two has been found in some studies,1 others indicate that vasomotor and neuropathic dysfunction develop independently.2 We have found support for the second view while comparing the results of a cold provocation test with electrophysiological nerve conduction velocity (NCV) studies.
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