Abstract
AbstractIt is well recognized that the kidney responds to sustained metabolic acidosis with a variety of metabolic adaptations. The metabolic pathways involved in renal ammonia formation, the potential rate-limiting steps, and the enzymatic modifications that accompany chronic metabolic acidosis have been the subject of several detailed reviews [1–4]. On the other hand, relatively little attention has been focused on the metabolic response which precedes the adaptations that characterize the chronic acidotic state, that is, the effect of acute acidosis or a low pH, per se, on renal metabolism.
Published Version
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