Abstract
Identifying individuals at risk and endophenotypes associated with adverse posttraumatic neuropsychiatric sequelae (APNS) vulnerability is of great significance for early interventions. Lower ventromedial prefrontal cortex (vmPFC) during inhibition paradigms has been associated with PTSD, but sample sizes were small and relevance to other APNS remained unclear. Here we examined whether vmPFC was specifically related to PTSD or reflect a transdiagnostic risk factor across APNS in a larger-scale prospective Emergency Department (ED) sample.
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