Reply to “Letter to the editor: ‘The role of short QT interval and elevated LV end-diastolic pressure in the genesis of ventricular tachycardia and fibrillation’”

Abstract

reply: We appreciate Dr. Karagueuzian's ([3][1]) insight regarding the potential role of reduced cytosolic ATP (derived from glucose and glycogen) in explaining shortening of the QT interval (through activation of the ATP-sensitive K+ channel) as well as elevations in end-diastolic pressure (through