Abstract
Esophageal duplication cysts are unknown lesions, with an approximate incidence of 1 in 10,000 [1]. They account for about 20 % of all gastrointestinal duplications [2]. Because of the reported association of esophageal duplications and esophageal atresia (EA) [3, 4], some authors have sought a possible commonality in the embryogenesis of these defects. When rats were given Adriamycin to develop EA and tracheoesophageal fistula (TEF), 25 % also developed esophageal duplications [5]. This finding implies that esophageal duplications arise from the foregut, and failure of the esophagus to normally separate from the notochord may contribute to their development. It has also been postulated that esophageal duplications and bronchogenic cysts share a common origin [6]. However, esophageal duplications are histologically distinct from bronchogenic cysts by having layers of smooth muscle and being lined by gastrointestinal epithelium. Most duplications are cystic and do not share a common wall with the esophagus.
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