Abstract
MOST physicians associate carbon tetrachloride poisoning with serious liver damage and jaundice. This is not surprising, since knowledge of this substance stems from medical-school teaching, in which carbon tetrachloride was cited as an etiologic agent in toxic cirrhosis or acute atrophy of the liver and emphasis was placed on the risk of liver damage following the improper use of carbon tetrachloride as a vermifuge. The association of carbon tetrachloride with liver injury is also due in part to familiarity with its use as a hepatotoxic agent in animal experiments.1 2 3 It is not generally recognized, however, that the inhalation of carbon . . .
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