Abstract
Exposure to acid-ethanol releases intense, previously masked insulin-like activity from the serum of both normal subjects and untreated diabetic patients. In proportion to the level of ILA in intact serum, diabetic serum contains less masked ILA than does normal serum. In terms of total ILA, however, that released from the serum of diabetics exceeds that released from normal serum, both in the fasting state and after glucose loading. The delayed increase of serum ILA after glucose loading, repeatedly observed in diabetics, has been shown to hold true for acid-alcohol releasable ILA as well. Exposure to acid-ethanol releases intense, previously masked insulin-like activity from the serum of both normal subjects and untreated diabetic patients. In proportion to the level of ILA in intact serum, diabetic serum contains less masked ILA than does normal serum. In terms of total ILA, however, that released from the serum of diabetics exceeds that released from normal serum, both in the fasting state and after glucose loading. The delayed increase of serum ILA after glucose loading, repeatedly observed in diabetics, has been shown to hold true for acid-alcohol releasable ILA as well.
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