Abstract
Hypercapnia in vagotomized cats can induce not only an increase in the depth of respiration, but also an increase in the frequency of inspiratory volleys. With deepening of anesthesia, the increase in depth of the inspiratory volleys continues, whereas the increase in the respiration rate disappears. These observations point to differences in the central mechanisms controlling the frequency and depth of respiration, of which the former is more susceptible to suprabulbar influences.
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