Abstract
One of the main causes for atopic dermatitis (AD) is skin barrier dysfunction which can be represented as increased trans-epidermal water loss (TEWL). Skin barrier dysfunction is induced by the disruption of stratum corneum (SC), a dense protein–lipid matrix which functions as a barrier to exogenous irritants and allergens. Recently a relationship between altered SC lipid profiles and impaired skin barrier function has been described [1,2]. SC lipids are composed of ceramides (CER), free fatty acids (FFA) and cholesterol (CHOL)[3].
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