Abstract
Montgomery1 and Monckeberg2 have observed altered levels of oxygen consumption by malnourished infants before and during therapy of nutritional deficits. There are indirect indications of changes in tissue oxygen metabolism in malnourished children. Glucose tolerance tests reveal delayed clearance of intravenous glucose loads3–4. It has been found that the malnourished infant has a reduced ability to utilize acetate, glucose and pyruvate when these were administered as 14C isotopes5.
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